Utilize este identificador para referenciar este registo: http://hdl.handle.net/10400.17/613
Título: Acute Bivalvular Left-Sided Methicillin-Resistant Staphylococcus Aureus Endocarditis with Cardiac, Cerebral, Renal and Septic Complications
Outros títulos: Endocardite Aguda Bi-Valvular Esquerda por Estafilococo Aureus Meticilino-Resistente com Complicações Cardíaca, Cerebral, Renal e Sepsis
Autor: Póvoas, D
Figueiredo, M
Murinello, A
Damásio, H
Ramos, A
Rodrigues, N
Sousa, J
Carvalho, F
Peres, H
Gomes, P
Palavras-chave: Doença Aguda
Lesão Renal Aguda
Válvula Aórtica
Bacteriemia
Endocardite Bacteriana
Resultado Fatal
Fungemia
Insuficiência Cardíaca
Estafilocócos Aureus Meticilino-Resistente
Infecções Relacionadas com Prótese
Infecções por Estafilocócos
Acidente Vascular Cerebral
AVC
HCC CAR
HCC MED
HCC NEF
HCC PAT CLIN
Data: 2011
Editora: Sociedade Portuguesa de Cardiologia
Citação: Rev Port Cardiol. 2011 Jun;30(6):611-20
Resumo: Infective endocarditis (IE) is now rare in developed countries, but its prevalence is higher in elderly patients with prosthetic valves, diabetes, renal impairment, or heart failure. An increase in health-care associated IE (HCAIE) has been observed due to invasive maneuvers (30% of cases). Methicillin-resistant Staphylococcus aureus (MRSA) and Enterococcus are the most common agents in HCAIE, causing high mortality and morbidity. We review complications of IE and its therapy, based on a patient with acute bivalvular left-sided MRSA IE and a prosthetic aortic valve, aggravated by congestive heart failure, stroke, acute immune complex glomerulonephritis, Candida parapsilosis fungémia and death probably due to Serratia marcescens sepsis. The HCAIE was assumed to be related to three temporally associated in-hospital interventions considered as possible initial etiological mechanisms: overcrowding in the hospital environment,iv quinolone therapy and red blood cell transfusion. Later in the clinical course,C. parapsilosis and S. marcescens septicemia were considered to be possible secondary etiological mechanisms of HCAIE.
Peer review: yes
URI: http://hdl.handle.net/10400.17/613
Aparece nas colecções:CAR - Artigos
PAT CLIN - Artigos
MED - Artigos
NEF - Artigos

Ficheiros deste registo:
Ficheiro Descrição TamanhoFormato 
Rev Port Card 2011 611.pdf222,25 kBAdobe PDFVer/Abrir


FacebookTwitterDeliciousLinkedInDiggGoogle BookmarksMySpace
Formato BibTex MendeleyEndnote Degois 

Todos os registos no repositório estão protegidos por leis de copyright, com todos os direitos reservados.